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But mutations in promoters sifted out of mountains of data by the team's novel mathematical formula, or algorithm, called GECCO, lay in genes never before implicated in pancreatic cancer.

Feigin points out that mutations in a promoter can affect how much protein is generated by the gene its regulates.

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lie adjacent to, but not within, the sequences of the genes that they regulate.

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But a new wave of research, exemplified by a study published in Nature Genetics by a team at Cold Spring Harbor Laboratory (CSHL), is significantly improving our ability to target cancer cells by studying "the other 98%" of DNA in human chromosomes, sometimes called the genome's "dark matter." Research led by Michael Feigin, Ph.

D., a postdoctoral researcher in the laboratory of CSHL Professor David Tuveson, M. D., looked closely at cells sampled from 308 people with pancreatic cancer, one of the most lethal malignancies, with a 5-year survival rate of only 8%. This enabled Feigin and colleagues including computational biologist Tyler Garvin, Ph.

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